Items where Author is "Hildyard, J C W"

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Number of items: 16.

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Verhaart, I E C and Cappellari, O and Tanganyika-de Winter, C L and Plomp, J J and Nnorom, S and Wells, K E and Hildyard, J C W and Bull, D and Aartsma-Rus, A and Wells, D J (2020) Simvastatin Treatment Does Not Ameliorate Muscle Pathophysiology in a Mouse Model for Duchenne Muscular Dystrophy. Journal of Neuromuscular Diseases. pp. 1-19. ISSN 2214-3602

Hildyard, J C W and Rawson, F and Wells, D J and Piercy, R J (2020) Multiplex in situ hybridization within a single transcript: RNAscope reveals dystrophin mRNA dynamics. PLOS ONE, 15 (9). e0239467. ISSN 1932-6203

Hildyard, J C W and Crawford, A H and Rawson, F and Riddell, D O and Harron, R C M and Piercy, R J (2020) Single-transcript multiplex in situ hybridisation reveals unique patterns of dystrophin isoform expression in the developing mammalian embryo. Wellcome Open Research, 5. p. 76. ISSN 2398-502X

Hildyard, J C W and Finch, A M and Wells, D J (2019) Identification of qPCR reference genes suitable for normalizing gene expression in the mdx mouse model of Duchenne muscular dystrophy. PLoS One, 14 (1). e0211384.

Amoasii, L and Hildyard, J C W and Li, H and Sanchez-Ortiz, E and Mireault, A and Caballero, D and Harron, R and Stathopoulou, T-R and Massey, C A and Shelton, J M and Bassel-Duby, R and Piercy, R J and Olson, E N (2018) Gene editing restores dystrophin expression in a canine model of Duchenne muscular dystrophy. SCIENCE, 362 (6410). pp. 86-91.

Hildyard, J C W and Taylor-Brown, F E and Massey, C and Wells, D J and Piercy, R J (2018) Determination of qPCR Reference Genes Suitable for Normalizing Gene Expression in a Canine Model of Duchenne Muscular Dystrophy. Journal of Neuromuscular Diseases, 5 (2). pp. 177-191.

Kondori, N R and Paul, P and Robbins, J P and Liu, K and Hildyard, J C W and Wells, D J and De Belleroche, J S (2018) Focus on the Role of D-serine and D-amino Acid Oxidase in Amyotrophic Lateral Sclerosis/Motor Neuron Disease (ALS). Frontiers in Molecular Biosciences, 5 (8).

Hildyard, J C W and Rawson, F and Harron, R and Riddell, D and Massey, C A and Taylor-Brown, F E and Wells, D J and Piercy, R J (2018) CHARACTERISING THE SKELETAL MUSCLE HISTOLOGICAL PHENOTYPE OF THE DELTAE50-MD DOG, A PRECLINICAL MODEL OF DUCHENNE MUSCULAR DYSTROPHY. Neuromuscular Disorders, 28. S18.

Kondori, N R and Paul, P and Robbins, J P and Liu, K and Hildyard, J C W and Wells, D J and De Belleroche, J S (2017) Characterisation of the pathogenic effects of the in vivo expression of an ALS-linked mutation in D-amino acid oxidase: Phenotype and loss of spinal cord motor neurons. PLoS One, 12. e0188912.

Hildyard, J C W and Lacey, E and Booler, H and Hopkinson, M and Wells, D J and Brown, S C (2016) Transgenic Rescue of the LARGEmyd Mouse: A LARGE Therapeutic Window? PLoS One, 11 (7). e0159853.

Hildyard, J C W and Wells, D J (2016) Investigating synthetic oligonucleotide targeting of miR31 in Duchenne muscular dystrophy. PLoS Currents.

Muses, S and Godfrey, C and McClorey, G and Wells, K E and Coursindel, T and Terry, R L and Betts, C and Cappellari, O and Hammond, S and O'Donovan, E and Hildyard, J C W and El Andaloussi, S and Gait, M and Wood, M and Wells, D J (2015) THE PHYSIOLOGICAL CONSEQUENCES OF DIFFERENT LEVELS OF DYSTROPHIN FOLLOWING ANTISENSE BASED EXON-SKIPPING IN THE MDX MOUSE. Neuromuscular Disorders, 25. S312-S313.

Godfrey, C and Muses, S and McClorey, G and Wells, K E and Coursindel, T and Terry, R L and Betts, C and Hammond, S and O'Donovan, L and Hildyard, J C W and El Andaloussi, S and Gait, M J and Wood, M J and Wells, D J (2015) How much dystrophin is enough: the physiological consequences of different levels of dystrophin in the mdx mouse. HUMAN MOLECULAR GENETICS, 24 (15). pp. 4225-4237.

Hildyard, J C W and Wells, D J (2014) Identification and validation of quantitative PCR reference genes suitable for normalising expression in normal and dystrophic cell culture models of myogenesis. PLoS Currents, 6.

Hildyard, J C W and Wells, D J (2014) Identification and validation of quantitative PCR reference genes suitable for normalizing expression in normal and dystrophic cell culture models of myogenesis.

Other

Hildyard, J C W The overexpression of LARGE in the LARGEmyd mouse restores the wild type phenotype despite a tissue specific variation in transgene levels. UNSPECIFIED.

This list was generated on Wed Dec 8 07:41:51 2021 GMT.